Search results for "Long-Chain Fatty"

showing 7 items of 7 documents

Taste of Fat: A Sixth Taste Modality?

2015

International audience; An attraction for palatable foods rich in lipids is shared by rodents and humans. Over the last decade, the mechanisms responsible for this specific eating behavior have been actively studied, and compelling evidence implicates a taste component in the orosensory detection of dietary lipids [i.e., long-chain fatty acids (LCFA)], in addition to textural, olfactory, and postingestive cues. The interactions between LCFA and specific receptors in taste bud cells (TBC) elicit physiological changes that affect both food intake and digestive functions. After a short overview of the gustatory pathway, this review brings together the key findings consistent with the existence…

0301 basic medicineFood intakeTastePhysiologyLong-Chain FattyAcid Transporter FatGlucagon-Like Peptide-1ReviewBiologyReceptors G-Protein-CoupledFood Preferences03 medical and health sciencesBud CellsRisk Factors2-Bottle Choice TestPhysiology (medical)Obesity-Resistant RatsAnimalsHumansGastric Bypass-SurgeryObesityGustatory pathwayTaste Bud CellsMolecular BiologyModality (semiotics)[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Fatty AcidsTaste PerceptionFeeding BehaviorGeneral MedicineTaste BudsDietary FatsSweet TasteVasoactive-Intestinal-Peptide030104 developmental biologyOverconsumptionBiochemistryTasteEating behaviorlipids (amino acids peptides and proteins)Digestive functionsReceptor-CellsNeuroscienceSignal Transduction
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Docosahexaenoic Acid Attenuates Mitochondrial Alterations and Oxidative Stress Leading to Cell Death Induced by Very Long-Chain Fatty Acids in a Mous…

2020

In the case of neurodegenerative pathologies, the therapeutic arsenal available is often directed towards the consequences of the disease. The purpose of this study is, therefore, to evaluate the ability of docosahexaenoic acid (DHA), a molecule present in certain foods and considered to have health benefits, to inhibit the cytotoxic effects of very long-chain fatty acids (C24:0, C26:0), which can contribute to the development of some neurodegenerative diseases. The effect of DHA (50 &micro

0301 basic medicineProgrammed cell deathDocosahexaenoic AcidsCell SurvivalVery long chain fatty acidoligodendrocytesvery long-chain fatty acidmedicine.disease_causeCatalysisArticleCell Linelcsh:ChemistryInorganic Chemistry03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicinemedicineAnimalsViability assayPropidium iodidePhysical and Theoretical Chemistrylcsh:QH301-705.5Molecular BiologySpectroscopyMembrane Potential MitochondrialOrganic ChemistryAutophagyFatty Acidsfood and beveragesGeneral Medicinelipotoxicitydocosahexaenoic acidComputer Science ApplicationsCell biologyMitochondriaOligodendrogliaOxidative Stress030104 developmental biologylcsh:Biology (General)lcsh:QD1-999chemistryLipotoxicityDocosahexaenoic acidModels Animallipids (amino acids peptides and proteins)Reactive Oxygen Species030217 neurology & neurosurgeryOxidative stressInternational Journal of Molecular Sciences
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LXR antagonists induce ABCD2 expression

2014

X-linked adrenoleukodystrophy (X-ALD) is a rare neurodegenerative disorder characterized by the accumulation of very-long-chain fatty acids resulting from a beta-oxidation defect. Oxidative stress and inflammation are also key components of the pathogenesis. X-ALD is caused by mutations in the ABCDI gene, which encodes for a peroxisomal half ABC transporter predicted to participate in the entry of VLCFA-CoA into the peroxisome, the unique site of their beta-oxidation. Two homologous peroxisomal ABC transporters, ABCD2 and ABCD3 have been proven to compensate for ABCD1 deficiency when overexpressed. Pharmacological induction of these target genes could therefore represent an alternative ther…

Agonistx-ald;very-long-chain fatty acid;lxr;hydroxycholesterol;abcd2medicine.medical_specialtymedicine.drug_classx-aldEndogenyContext (language use)ATP-binding cassette transporterBiologyATP Binding Cassette Transporter Subfamily DInternal medicinemedicineHumanslxr[ SDV.BDD ] Life Sciences [q-bio]/Development BiologyhydroxycholesterolLiver X receptorAdrenoleukodystrophyMolecular Biology[SDV.BDD]Life Sciences [q-bio]/Development BiologyLiver X ReceptorsFatty AcidsBiologie du développementNeurosciencesCell BiologyHep G2 CellsPeroxisomemedicine.diseaseOrphan Nuclear ReceptorsDevelopment BiologyHydroxycholesterolsvery-long-chain fatty acidOxidative StressEndocrinologyGene Expression RegulationCell cultureabcd2Neurons and Cognition[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Cancer researchlipids (amino acids peptides and proteins)AdrenoleukodystrophyATP-Binding Cassette Transporters[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]
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Obesity alters the gustatory perception of lipids in the mouse: plausible involvement of lingual CD36. : Obesity decreases the fat preference

2013

International audience; A relationship between orosensory detection of dietary lipids, regulation of fat intake, and body mass index was recently suggested. However, involved mechanisms are poorly understood. Moreover, whether obesity can directly modulate preference for fatty foods remains unknown. To address this question, exploration of the oral lipid sensing system was undertaken in diet-induced obese (DIO) mice. By using a combination of biochemical, physiological, and behavioral approaches, we found that i) the attraction for lipids is decreased in obese mice, ii) this behavioral change has an orosensory origin, iii) it is reversed in calorie-restricted DIO mice, revealing an inverse …

CD36 AntigensCD36[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionAdipose tissueMESH : Behavior AnimalBiochemistryCalcium in biologyMice0302 clinical medicineEndocrinologyMESH : Calcium SignalingMESH: Behavior AnimalMESH: ObesityMESH: AnimalsLingual papillaResearch Articles2. Zero hunger0303 health sciencesMESH : Food PreferencesBehavior AnimalMESH : TongueMESH : Diet High-FatMESH: TongueTaste Perceptiontaste sensitivityMESH : Antigens CD36calcium imagingAdipose TissueHealthMESH: Dietary FatsMESH : ObesityFat tasteMESH: Adipose Tissuemedicine.medical_specialtyFood behavior030209 endocrinology & metabolismMESH : Mice Inbred C57BLQD415-436BiologyDiet High-FatMESH: Calcium SignalingMESH : Adipose TissueFood Preferences03 medical and health sciencesCalcium imagingTongueDownregulation and upregulationMESH: Mice Inbred C57BLInternal medicineMESH : MicemedicineAnimalsCalcium SignalingObesityFatty acidsMESH: Food PreferencesMESH: Mice030304 developmental biologyNutritionlong-chain fatty acidsMESH: Antigens CD36MESH : Taste PerceptionCell Biologymedicine.diseaseDietary FatsObesityMice Inbred C57BLMESH: Diet High-FatEndocrinologyMESH: Taste Perceptionbiology.proteinMESH : AnimalsBody mass index[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH : Dietary Fats
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Lipid-mediated release of GLP-1 by mouse taste buds from circumvallate papillae: putative involvement of GPR120 and impact on taste sensitivity

2012

Glucagon-like peptide-1 (GLP-1) signaling modulates sweet-taste sensitivity in the mouse. Because circumvallate papillae (CVPs) express both GLP-1 and its receptor, a local regulation has been suggested. However, whether dietary lipids are involved in this regulation, as shown in the gut, is unknown. By using a combination of biochemical, immunohistochemical, and behavioral approaches, the present data i) confirm the role of GLP-1 signaling in the attraction for sucrose, ii) demonstrate that minute quantities of long-chain FAs (LCFAs) reinforce the attraction for sucrose in a GLP-1 receptor-dependent manner, iii) suggest an involvement of the LCFA receptor GPR120 expressed in taste buds in …

CD36 Antigensmedicine.medical_specialtyTasteendocrine systemCD36Blotting WesternQD415-436eating behaviorReal-Time Polymerase Chain ReactionBiochemistryGlucagon-Like Peptide-1 ReceptorReceptors G-Protein-CoupledMiceEndocrinologyTAS1R3TAS1R2Glucagon-Like Peptide 1Cell Line TumorInternal medicinelong-chain fatty acidReceptors GlucagonmedicineAnimalsHumansSecretionObesityReceptorLingual papillaResearch Articlesbiologydigestive oral and skin physiologyGPR120healthCell BiologyTaste BudsImmunohistochemistryMice Inbred C57BLEndocrinologyobesity riskbiology.proteinJournal of Lipid Research
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Long-chain fatty alcohols from pomace olive oil modulate the release of proinflammatory mediators

2009

Pomace olive oil is a by-product of olive oil extraction that is traditionally produced and consumed in Spain. The nonglyceride matter of this oil is a good source of interesting minor compounds, like long-chain fatty alcohols, which are present free or as part of waxes. In the present study, long-chain fatty alcohols were isolated from the nonglyceride fraction of pomace olive oil, and the composition was identified and quantified. The major components of long-chain fatty alcohols were tetracosanol, hexacosanol and octacosanol. We investigated the ability of long-chain fatty alcohols from pomace olive oil to inhibit the release of different proinflammatory mediators in vitro by cells invol…

Cell SurvivalNeutrophilsEndocrinology Diabetes and MetabolismClinical BiochemistryNitric Oxide Synthase Type IIBiochemistryProinflammatory cytokineMiceAnimalsPlant OilsPomace olive oilPhospholipases A2 SecretoryMolecular BiologyOlive OilCytokineCalcimycinInflammationNutrition and DieteticsChemistryMacrophagesPomaceNitric oxideRatsThromboxane B2BiochemistryLong-chain fatty alcoholsFatty AlcoholsInflammation MediatorsLong chainOlive oil
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Taste for fat: the 6th taste modality

2016

IF 4.066; International audience

Vasoactive-Intestinal-PeptideBud Cells[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]2-Bottle Choice Test[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Obesity-Resistant RatsLong-Chain FattyAcid Transporter FatGlucagon-Like Peptide-1Gastric Bypass-SurgeryReceptor-CellsSweet Taste
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